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Uraemic cardiomyopathy is characterized by extensive metabolic remodelling, including secondary carnitine deficiency, reduced fatty acid use, enhanced glucose utilization, mitochondrial dysfunction and altered expression of metabolic regulatory genes, which collectively result in impaired energetics

Peptides are short chains of amino acids, the building blocks of proteins

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Delivery of cells and drugs to the injury site and their long-term maintenance at the site is critical for accelerated target organ repair and functional recovery